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Kansuinine E
CAS No. 672945-84-5
Kansuinine E( —— )
Catalog No. M31347 CAS No. 672945-84-5
Kansuinine E, a jatrophane-type diterpenoid, is a plant-derived compound isolated from the roots of E. kansui. It functions as a nitric oxide inhibitor, with an inhibitory concentration of 6.3 μM (IC50).
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 5MG | 873 | In Stock |
|
| 50MG | Get Quote | In Stock |
|
| 100MG | Get Quote | In Stock |
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Biological Information
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Product NameKansuinine E
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NoteResearch use only, not for human use.
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Brief DescriptionKansuinine E, a jatrophane-type diterpenoid, is a plant-derived compound isolated from the roots of E. kansui. It functions as a nitric oxide inhibitor, with an inhibitory concentration of 6.3 μM (IC50).
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DescriptionKansuinine E, a jatrophane-type diterpenoid, is a plant-derived compound isolated from the roots of E. kansui. It functions as a nitric oxide inhibitor, with an inhibitory concentration of 6.3 μM (IC50).
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In Vitro——
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In Vivo——
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Synonyms——
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PathwayOthers
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TargetOther Targets
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Recptor——
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Research Area——
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Indication——
Chemical Information
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CAS Number672945-84-5
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Formula Weight777.8
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Molecular FormulaC41H47NO14
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Purity>98% (HPLC)
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Solubility——
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SMILES——
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Chemical Name——
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
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Dodecyl isobutyrate
Dodecyl isobutyrate (Dodecyl 2-methylpropanoate) is a compound found in Jatropha curcas that causes an escape reaction in thrips.
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[D-Trp11]-Neurotensi...
[D-Trp11]-Neurotensin, an analogue of Neurotensin (NT), is a selective antagonist of NT in perfused rat hearts but behaves as a full agonist in guinea pig atria and rat stomach strips. [D-Trp11]-Neurotensin can inhibit NT-induced hypotension.
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N'-Methylnicotinamid...
N-methylnicotinamide is a metabolite of niacin (or nicotinamide) and is commonly found in human urine. However low levels of urinary excretion of N-methylnicotinamide indicates niacin deficiency. In patients with liver cirrhosis nicotinamide methylation is increased leading to a rise in urinary N-methylnicotinamide. N-methylnicotinamide has been found to be a microbial metabolite.
